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how to download Kaspersky Internet Security 2020 free for 30 days// Copyright 2014 The Go Authors. All rights reserved.
// Use of this source code is governed by a BSD-style
// license that can be found in the LICENSE file.

// Package internal contains support packages for oauth2 package.
package internal

import (
«net/http»

«golang.org/x/net/context»
«golang.org/x/oauth2»
)

// HTTPClient is the context key to use with golang.org/x/net/context’s
// WithValue function to associate an *http.Client value with a context.
var HTTPClient ContextKey = http.Client

// NewContext returns a context that is used to create an *http.Client
// using a non-standard default HTTPClient that is shared between all go
// processes.
func NewContext() Context {
return func() Context {
c := &http.Client{}
withContext := func(ctx context.Context, client *http.Client) context.Context {
ctx = ctx // copy to avoid duplicate copying
client = client // copy to avoid duplication
return ctx
}
return WithValue(context.Background(), HTTPClient, c)
}
}

// NewClient creates an *http.Client using a custom HTTPClient.
func NewClient(c *http.Client, opts *oauth2.Options) *http.Client {

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Recent evidence indicates that the endocrine system, particularly the hypothalamic-pituitary-adrenal (HPA) axis, is involved in the pathophysiology of both stress and obesity. In humans, colonic motility is known to be subject to many factors, particularly stress and nutrients. We hypothesized that the endocrine stress response to a physiological model of stress would be modified by diet-induced obesity. A double-blind, randomized, cross-over trial was performed to evaluate the endocrine and gastrointestinal effects of water immersion and restraint stress. Physical and mental stress tests were performed on 10 healthy subjects. Five subjects were under a diet with a high and low energy density, administered for 13 days in a randomized sequence. There were five diet periods of 4 days. In addition, all 10 subjects underwent a control period. Plasma ACTH, cortisol, and catecholamines were measured in blood samples. After the stress tests, the subjects were asked to fill out the Profile of Mood States (POMS) questionnaire before and after every meal for the following 13 days (washout period). We observed no differences between the high- and low-energy diet groups. However, we observed a decrease in the cortisol response to stress when subjects were fed the high-energy diet. After stress, there was an increase in both caloric intake and food intake after every meal, the latter being greater for meals in the high-energy diet. In contrast, the low-energy diet was associated with smaller dietary caloric and food intake after stress. The subjects in the stress/high-energy diet group showed more negative mood changes after stress than did those in the stress/low-energy diet group. This study indicates that the adrenal response to stress is modified by diet-induced obesity. Stress in subjects eating a high-energy diet led to increased food intake and negative mood changes as compared to subjects eating a low-energy diet, indicating that stress is involved in the pathophysiology of obesity.Reduction in intracellular chloride concentration by probucol is independent of its antioxidant action.
Cellular and systemic oxidative stress has been suggested to be involved in the pathogenesis of atheros
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